By Leopold G. Koss and Dulcie V. Coleman (Auth.)
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Extra info for Advances in Clinical Cytology
Microfilaments (arrow) are visible even in the most poorly differentiated gland cells of endometrial origin. (x 6,800) 34 (a) * t t (b) I - ■v 4 I Α**^Κ ^JT Alex Ferenczy 35 decreased1114. Further involution of the epithelium occurs by autophagy, single total cell deletion (apoptosis) and coagulative necrosis11,14. 6). The elevated acid phosphatase content of progestogen-treated endometrial adenocarcinoma in the human22 and the prominent autophagic activity observed in rat uterine adenocarcinoma cells cultured and treated with P in vitro54 are also reminiscent of the changes seen in premenstrual secretory endometrium.
D. and HAHNEL, R. (1974) Estrogen receptor concentration in normal and pathological human uterine tissues, Journal of Clinical Endocrinology and Metabolism, 38, 23-32 2 6 . , KREUZER, G. a n d POLLOW, B. (1975) Characterization and comparison of receptors for 17ß estradiol and progesterone in human proliferative endometrium and endometrial carcinoma, Endocrinology, 26, 319-328 27. , GUSBERG, s. B. and GURPIDE, E. (1977) Estradiol receptor and 17ß dehydrogenase in normal and abnormal human endometrium, Annals of the New York Academy Sciences, 286, 190-198 28.
The glandular epithelium is thicker than normal and is often multilayered. The epithelial cells often have a slightly increased nucleocytoplasmic ratio. The stroma is abundant and cellular, and mitotic figures are numerous in both glands and stroma (see Plate 1). Adenomatous hyperplasia In this disorder the glandular epithelium is hyperplastic and the stroma is not usually as conspicuous as in cystic hyperplasia, so that in the most severe forms the stroma appears to be almost completely excluded.
Advances in Clinical Cytology by Leopold G. Koss and Dulcie V. Coleman (Auth.)